The Treatment of Stage IV Cancers Chapter 8:
Dental Issues – Part 2
Written by
Webster Kehr, Independent Cancer Research Foundation, Inc. | Last updated on February 17, 2014 | Filed under: Cancer Articles,
Conspiracy
Theories, Treatments
Chapter 8: Dental Issues – Part 2
By Suzin Stockton
Used by Permission of the Author from the Y2K Health and Detox Center
Used by Permission of the Author from the Y2K Health and Detox Center
JAWBONE CAVITATIONS: Infarction, Infection & Systemic Disease
About
seven years ago I made the eye-opening discovery that my chronic health
problems, which had eluded resolution for many years, had their origin in – of
all places – my jawbone. I would never have deduced this had it not been for a
fortuitous chance finding: the complete disappearance of bladder problems of
one-year duration within days of having an abscessed tooth removed.
When
I reported the “coincidence” to my dentist, he was incredulous. He shouldn’t
have been, for he considered himself to be a “holistic” practitioner. That same
dentist would later, despite his lack of understanding of focal illness, unknowingly
help me to learn more about it by creating the conditions that triggered the
full expression of a long silent jawbone disease – ischemic osteonecrosis.
(a.k.a. cavitations and a dozen or so other names). This disease is actually
quite common, though infrequently diagnosed, and is perhaps THE most common
focal condition in the body. A “focus” is a walled-off area of concentrated
toxins and necrotic (dead) and/or infected tissue.
Ischemic
osteonecrosis (bone death due to poor blood supply) is a disease of the entire
skeleton – i.e., it can affect any bone in the body. It is best known as a hip
condition, and yet it is actually more common in the jawbone, though
unacknowledged as such by mainstream medicine and dentistry.
A
jawbone cavitation is simply a hollow space or pocket in the bone. It is not
readily visible to the eye and often causes no local discomfort, though it can
be the hidden cause of facial pain syndromes (hence one of its names, NICO –
Neuralgia Inducing Cavitational Osteonecrosis). The chief initiating factor is
trauma to the jaw, often brought on by standard dental treatment.
I’d
first encountered the word, “cavitation” many years ago in the writings of Dr.
Hulda Clark. She’d described it in her books as “a bone infection resulting from
an incompletely extracted tooth” – i.e., an extraction where tissue (bone and
ligament) that should be completely extracted is not thoroughly removed. That
description didn’t resonate in me then, despite the fact that it was exactly
what had been silently going on in my jawbone for many years. I guess I thought
if I had an infection in my jaw, I’d know it: Surely there would be pain,
inflammation, tenderness – and my dentist would find the problem in the course
of my routine check-ups. WRONG!
Chronic
osteomyelitis (cavitation) of the jawbone is not characterized by the usual
signs of infection (inflammation, redness, fever, pus) – it most often is a
silent condition. And it’s one that dentists are not trained in school to
recognize. In fact, they’re not even taught that the condition exists. This is
a somewhat perturbing state of affairs, for the jawbone cavitation is not a new
disease.
It
was described as early as 1848 by Thomas Bond in the first oral pathology book.
He wrote about a jawbone necrosis that existed independently of abscessed teeth
and gums. In 1915, Dr. G.V. Black, the father of modern dentistry, described
the condition as “chronic osteitis.”
Jawbone
cavitations are exquisitely described in an eye-opening book entitled Death and Dentistry written in 1940 by
Martin H. Fischer, medical doctor and professor of physiology at the University
of Cincinnati. Citing the research of Drs. Frank Billings and E.C. Rosenow
(early 1900s), Dr. Fischer speaks of “infarctions induced of microorganismal
emboli” that have broken into the general circulation from a peripheral focal
point in the jaw or tonsils. This “metastasis” of microorganisms is the cause
of a surprising number of conditions according to Fischer (p.8, 9):
Embolic
infection that has struck the heart valves will be endocarditis; the heart
muscle, myocarditis; the pericardium, pericarditis; if all are struck, it is
pancarditis. Involving the skeletal muscles, the same pathological background
will give rise to myositis; when their tendinous junctions are struck,
fibrositis; and when the synovial bursae are affected, bursitis or
tenosynovitis. The process in the joints is arthritis; and in the nerves and
nerve ganglia, neuritis. In the brain, this is cerebritis, and in its
coverings, meningitis.
Death and Dentistry, by Martin H. Fischer, pages 8-9
Fischer,
Martin H., Death and Dentistry. Charles C. Thomas, LTD:
Springfield,
IL, 1940.
Fischer
goes on to explain the role of metastatic infection in gastric and duodenal
ulcers, cholecystitis, cystitis, pneumonia, bronchitis, rheumatism, asthma,
pleuritis, nephritis, thyroid disease, herpes, iritis, poliomyelitis, multiple
sclerosis, certain skin disorders, diabetes, migraines, hypertension and more.
He gives case histories and much clinical and laboratory evidence, including
impressive photographs of cross-sections of infected teeth and microscopy
slides.
Although
infection in the oral cavity may be a triggering event in the formation of a
cavitation, biopsy of the site typically shows few, if any, bacteria. It is the
toxins produced by these anaerobic bacteria that are most damaging to the body.
However, until local defenses break down and these toxins gain systemic access,
the problem remains localized and most likely silent.
Symptoms
develop when the body burden of toxins increases to the point that nutritional
reserves are depleted, and the system is no longer able to confine the toxins
to their point of origin. They then travel via blood and lymph channels and
through nerve pathways to other areas of the body.
Toxins
create an extremely acidic environment. As long as the body’s alkaline reserves
(primarily calcium and sodium) remain intact, pH is kept within acceptable
limits, homeostasis remains intact, and the body functions normally. Once
alkaline reserves are depleted however, balance is disrupted. It is not only
acid-forming foods (like grains and meat) so prevalent in the standard American
diet, that deplete the alkaline reserves, but also the bacterial toxins
generated at the site of jawbone cavitations.
These
toxins create an acid environment and destroy critical enzyme systems in the
body, including enzymes essential for energy production. The inactivated
enzymes are then unable to fulfill their function as mineral chaperones. The
net result is that key minerals, even though present in the system, become
bio-unavailable, for the enzymes needed to activate them have been destroyed by
bacterial toxins.
It
is important to understand that such a mineral “deficiency” is unrelated to
mineral intake. It can exist in the face of ample intake, though insufficient
intake certainly compounds the problem. The toxins responsible for mineral
deactivation and breakdown of homeostasis are carried throughout the system via
blood and lymph vessels, tending to settle in areas of inherent or acquired
weakness.
This
means that my jawbone cavitations may result in an entirely different symptom
picture than yours, simply because my weaknesses are different than yours.
The
over-acid conditions that result once alkaline reserves are depleted have many
deleterious systemic effects. When the pH of the blood becomes too acid, its
viscosity increases – that is to say it becomes thicker. Consequently, it does
not flow as smoothly through the vessels as it once did. Clotting anomalies
result. A tendency to excessive clotting is very common in chronic cavitation
patients, affecting approximately 80% of them. Hyper coagulation leads to
infarctions in blood vessels.
Jawbone
infarctions were spoken of by Dr. Fischer more than half a century ago.
Although the word, “infarct” has come to be associated with heart attack, the
condition is not confined to the large vessels associated with the heart.
Webster defines an “infarct” as “an area of necrosis in a tissue or organ
resulting from obstruction of the local circulation by a thrombus or embolus.”
Jawbone
necrosis does indeed result from impeded circulation, commonly stemming from
trauma to the jawbone. Such trauma is largely iatrogenic, the result of
standard dental treatment. Any large fillings, crowns, bridges (including the
once healthy teeth used as abutments for the bridge) veneers, endodontic
treatment, periodontal scaling, tooth extractions, injections (particularly of
vasoconstrictive anesthetics), placement of toxic and/or incompatible
restorative materials – all of these insults to the jawbone seriously reduce
the blood supply to it. Where blood supply is compromised, toxins can’t get
out, nutrients and oxygen can’t get in.
By
the time toxins gain systemic access, alkaline reserves have become depleted.
The blood then becomes hyper viscous, and infarction can occur. Such infarction
tends to occur initially in the small vessels associated with traumatized bone
tissue in the jaw. These infarctions of the microcirculation, it would appear,
are a major factor in the development and spreading of jawbone cavitations.
Fischer understood this years ago when he wrote of “infarctions induced of
microorganismal emboli.”
The
dental trauma most often associated with cavitations is the standard tooth
extraction, particularly if it involves the third molar (or wisdom tooth)
sites. Although taught in dental school, it is not common practice today for
the surgeon excavating these teeth to thoroughly remove the periodontal
ligament that attaches tooth to bone.
Once
the tooth is removed, this ligament serves no purpose, and if any part of it is
permitted to remain in the jaw, it serves as a barrier to healing, impeding
blood flow and preventing re-growth of bone. While the extraction site will
invariably “heal” shut, the healing is quite often incomplete, for below the
healed-over surface, a pocket or hole has formed. This hollow space becomes a
breeding ground for anaerobic microorganisms.
It
is very possibly these microorganisms that form the infarction-inducing embolus
of which Fischer wrote so many years ago. When the metabolic waste products of
these bacteria interact with chemical toxins (from restorative materials,
anesthetics, etc.) in the oral cavity, the result is the production of super
toxins. The extreme toxicity thus created may well reduce bacterial population.
Whether
or not a cavitation forms following the standard extraction of a tooth will
depend largely upon how much of the periodontal ligament happens to be removed
with the tooth (some portion usually comes out, even when the surgeon is making
no attempt at removal of it) AND the type of microorganisms which are present
at the site.
More
damaging than the microorganisms themselves are the extremely potent toxins
they produce. Once these bacterial toxins gain systemic access, they can do a
great deal of harm through inhibition of enzymes and minerals as described
above. The necrosis they produce is actually a gangrenous condition, which
tends to spread to other areas of the jawbone. Detoxification is a significant
challenge at this point and an absolute impossibility in the face of the
continuance of the focal condition (infected tooth and/or jawbone).
Treatment
of choice for jawbone cavitations is surgical removal of the necrotic and
infected bone, for in the presence of such bone, the conditions that created
the infection remain, and blood supply continues to be impaired. This surgical
procedure is a relatively simple one when done in conjunction with a new
extraction.
It
is much more difficult where old extraction sites are concerned. Here the task
is complicated by the fact that there has been, up until very recently, no way
to clearly visualize the cavitation site and gain information about its
dimensions and other distinguishing features short of opening up the site and
‘looking around.’ Even then, the site cannot be viewed from all angles.
To
the trained eye, the panoramic x-ray can reveal indications of the presence of
a cavitation, but not always. Even when it does, details are often not clearly
discernible, and the surgeon is still operating ‘in the blind’ to some degree.
The 2-dimensional x-ray image cannot adequately reflect anomalies in the
3-dimensional jawbone. In some instances, cavitations can be depicted on x-ray;
however, as much as 50% of the bone must be affected before their presence is
apparent.
The
MRI, while the ‘gold standard’ for detecting osteonecrosis of the hip, does not
work well with the flat bones of the face. Tech 99 bone scans are about 70%
effective when a special contrast medium is used. Jawbone cavitations can also
be imaged through CT scan, when a spiral scan is taken from about the middle of
the sinus to the bottom of the mandible.
These
methods, however, are neither practical nor cost-effective for use by the
dental profession. They expose the patient to the adverse effects of radiation
and require the interpretive services of a radiologist who is unlikely to
recognize jawbone cavitations because he has not been trained to do so. The
aware dentist has long been in need of a reliable instrument for clearly and
safely imaging jawbone cavitations; ideally an instrument that could be used
“in house.” Such an instrument is now available, due to the unflagging efforts
of Bob Jones. The story of his dental drama is interesting, more dramatic than
my own (told in my book, Beyond Amalgam) and worth telling here.
A
decade ago, Bob was a specimen of perfect health – or so it seemed. He was
employed full-time as a commercial airline pilot, worked part-time as a ski
instructor. This avid outdoorsman was slim, trim and fit. That all changed in
1987 when he was stricken with chronic debilitating fatigue, muscle atrophy and
a neurological condition that baffled specialists.
By
1992, he had become completely disabled, was wheelchair bound, had lost use of
his arms and gained an excessive amount of weight. While the MDs couldn’t come
to agreement on the exact nature of the problem and finally settled upon a
speculative diagnosis of ALS, they were in agreement on one thing: Bob’s condition
was terminal.
They
had given him no more than six months to live, when he stumbled upon an
understanding of the source of his problem and a way to turn it around. His
search for solutions led him to the realization that potent toxins, by-products
of standard dental treatments were essentially poisoning his system. Bob’s
symptoms subsided, and his condition dramatically improved once his diseased
bone marrow and “silver” fillings were removed.
Today
he is completely mobile and moderately active. Much of his excess weight has
been lost. Bob is quick to point out, however, that his recovery has not been
100%. At this point in time, chronic cavitation patients can expect improvement
but often not complete cure, owing to the severity and duration of their
condition.
Even
before his recovery, Bob set out to develop an instrument designed to detect
jawbone cavitations. Since these lesions routinely elude detection through
standard diagnostic procedures, the need for an improved imaging device was
apparent. As a design engineer with a background in sonar technology, Bob was
convinced from the onset that such an imaging device could be developed using
sonography. Six months after commencing the arduous task of ‘cleaning out’ his
jawbone, Bob had developed the first working prototype of the CAVITAT™. There
would be many design revisions and obstacles put in his path in the years to
follow, but he worked diligently to make his vision of a perfected CAVITAT™ the
reality that it has now become.
The
CAVITAT’s proprietary analog to digital circuitry has been awarded 19 patents.
There are 22 additional patents pending on the flexible circuit receiver and
its advanced cross-channel noise suppression technique. The device is unique in
the sonography market in that it is engineered to show only bone, no soft
tissue. All other ultrasound devices do just the opposite – show tissue but no bone.
And, the image they display is 2-dimensional, while the CAVITAT™ displays a
3-dimensional color-coded image.
These
colors (green, yellow, red) reflect the degree of bone loss and necrosis. The
3-D computer images may be rotated so that they can be viewed from all angles.
One image is generated for each of the 32 tooth sites, and all can be displayed
on the screen simultaneously. This allows the operator to see the overall
picture and how one affected site can influence adjacent ones. Each of the 32 images
consists of 64 elements or pixels. These detailed images are identified as to
orientation – “B” for buccal and “D” for distal.
The
new Generation 4 CAVITAT™ differs from its prototype precursor in many
important respects. The resolution has been increased 800%, making for a much
clearer image and enabling detection of smaller cavitations. The Generation 4
is capable of detecting jawbone defects down to 1/64 of an inch in diameter.
Bob
Jones had introduced a limited number of Generation 3 CAVITATs to a select
number of dentists at the end of 1999. These were prototype models used for
field evaluation. The feedback from the dentists using them provided the data
necessary to make desired improvements. The software was totally rewritten, and
the net result was a user-friendly state-of-the-art precision instrument. It is
this version of the CAVITAT™ that is now being made available to doctors and
dentists to assist in diagnosis of jawbone cavitations and other bony defects
of the jaw.
The
significance of this technological break through cannot be overemphasized. The
success of cavitation surgery is dependent upon many variables. A major one is
the extent to which necrotic tissue is removed. Before the advent of the
CAVITAT™, dentists were operating very much in the blind, unable to see the
full extent of the necrosis and therefore unable to remove all necrotic bone.
The result for many patients was poor bone healing, unchecked spreading of
necrotic lesions and consequent need for repeat surgeries.
While
excision of all diseased bone will not necessarily assure full recovery, it
certainly does improve the odds. Most patients have had jawbone cavitations for
a number of years before they are discovered. Consequently, by the time
treatment is initiated, a great deal of serious damage has been done.
Dr.
Fischer had stated in Death and
Dentistry, “It is only in the earliest stages of oral disease that arrest
of progressive infection seems possible.” With the development of the CAVITAT™,
early detection is finally possible. It may be our only hope of putting the
reigns on this silent, insidious condition that appears to have reached
epidemic proportions.
While
thorough excision of osteonecrotic lesions is necessary in the treatment of
cavitations, for the chronic cavitation patient, it is often not sufficient.
Aggressive detoxification measures are also in order. These must be tailored to
the needs of the individual patient with regard to his/her specific
detoxification capabilities and overall condition. Nutritional support is also
essential – for rebuilding bone, improving circulation, combating infection,
chelating heavy metals.
While
surgical treatment of cavitations falls within the domain of the dental
profession, the metastatic infection seeded by these lesions has systemic
consequences that should be of interest to all physicians. It is therefore
imperative that every patient history taken by all physicians and health care
providers include questions about dental treatment. Remember: Any trauma to the
jaw can be the beginning of cavitations.
The
high-speed drill routinely used by dentists cracks enamel, thus allowing
bacterial toxins to penetrate the dentine. There is evidence that such drills
cause actual pulp damage. Drilling done then in preparation of a tooth for
routine fillings, crowns and bridges can be damaging to the jawbone.
Root
canals will unquestionably cause cavitations sooner or later, as will routine
extractions (where the socket is not properly cleaned out, with all
necrotic/infected bone removed). The eclectic physician will not only want to
question his patients about these procedures, s/he will also want to be in a
position to diagnose jawbone cavitations, or to refer patients to a dentist who
is able to make such a diagnosis. Once the diagnosis is made, it is desirable
that the dentist and primary physician work together in instigating a treatment
plan and following up with patient.
In
working with the chronic cavitation patient, it is imperative that the entire
jawbone be considered and examined – not just the site(s) of extractions. A
mistake that is frequently made is to clean out new extraction sites, while
ignoring old ones. If all necrosis is not removed, it will spread – and will
ultimately re-infect a new extraction site, even one that was properly cleaned
out.
Taking
things a step further, it is important to be aware that the spreading of
jawbone cavitations is not confined to edentulous areas. When the bone beneath
an apparently “vital” tooth becomes affected/infected, blood supply to that tooth
is greatly reduced, and it begins to die. Neither oral exam, nor x-ray
evaluation will likely reveal a problem with such a tooth.
ElectroDermal
Screening and muscle testing may also miss the problem. The patient, however,
frequently has a sense of something being “not quite right” with the tooth.
(The chronically sensitive tooth often is an indication of the presence of
jawbone necrosis beneath it) If he or she insists upon its extraction (usually
against the advice of the dentist) and manages to talk his/her dentist into
removing it, that dentist is counseled to carefully examine the extracted
tooth.
Chances
are very good that upon drilling into the pulp chamber, s/he will find that the
tooth is dead or dying. This avitality is reflected by lack of moisture in the
pulp chamber, a result of severely restricted blood flow. I say all of this
from personal experience, for three of my mandibular extractions done in ’99
and ’00 were performed at my insistence against the initial protestations of my
dentist, who fortunately was open-minded and curious enough to drill open the
pulp chambers of the extracted teeth.
Dentists
are taught to save the tooth at all costs. Frequently, however, the price paid
is the systemic health of the patient. Dead and dying teeth should not remain
in the jaw, even if they are causing no acute distress to the patient. If
CAVITAT™ scan of the jawbone shows pronounced necrosis under a “vital” tooth,
please entertain the possibility that the tooth only appears to be vital, and
is, in fact, dying. Healthy teeth don’t grow out of necrotic bone.
For
the chronic cavitation patient, extraction may be both the beginning and end of
his or her health problems. The improperly done extraction (usually of a wisdom
tooth) is frequently the beginning of a problem which may go undetected for
decades, and then only be resolved by the proper extraction of some, or
possibly all, of the remaining teeth, along with removal of necrotic bone from
edentulous areas and aggressive systemic detoxification.
Prevention
and early detection are the keys to avoiding this outcome. Improved imaging
capabilities give us the tool for such early intervention.. The first step in
solving the problem, however, is awareness of it. You have taken that step and
are urged to take the next one.
Doctors:
Learn to recognize jawbone cavitations and to either treat them surgically, or
refer your patient to a qualified cavitation surgeon for treatment. Patients:
Seek out a dentist familiar with jawbone pathology: It may be the unsuspected
cause of your systemic problems.
Note by Cancer Tutor: So what does all of this have to do
with cancer? Many cancer cases, as reported by Bill Henderson, can NEVER be cured unless the dental issues
discussed above and in the prior chapter are corrected.
The
reason is that cancer is caused by a very special highly pleomorphic, cell-wall
deficient bacteria which can be found inside of every cancer cell. These
microbes block the production of ATP molecules in cancer cells in several
different ways. The microbe which causes cancer can “hide” in the mouth. This
microbe, and the massive toxins caused by dental procedures, can continually
reinfect the cancer.
However,
not all cancer patients need the $10,000 or more procedures to fix their dental
issues and finally cure their cancer; though for those who can afford it, it is
certainly a highly recommended procedure for general health, if not for cancer.
Breast
cancer patients are almost certainly in need of these procedures, and should
automatically have them done, but any type of cancer can be caused by these
procedures. See:
Mike
Vrentas, who developed the Cellect-Budwig protocol, has developed a special
expertise with regards to breast cancer cases and is highly recommended as a
telephone consultant even if the Cellect-Budwig protocol is not used.
The
individual dentists who do this procedure have largely gone “underground.” This
does not mean they have fled to Mexico, it simply means that they must “hide in
the grass” and avoid too much exposure. But they can be found.
It
should also be emphasized that “biological dentists” and “holistic dentists”
are generally NOT qualified to deal
with the dental issues mentioned above!!
Ways
to find the very rare dentists who are qualified was mentioned in the prior
chapter.
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Article Topics
- Cavitat
- Death and Dentistry
- Infarction
- Jawbone Cavitations
- Martin H. Fishcer
- Osteonecrosis
- Root Canals
- Suzin Stockton
- Toxins
- Y2K Health and Detox Center
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