The Treatment of Stage IV Cancers Chapter 8:
Dental Issues – Part 2
Written by Webster Kehr, Independent Cancer Research
Foundation, Inc. | Last updated on February 17, 2014
| Filed under: Cancer Articles, Conspiracy Theories, Treatments
Chapter 8: Dental Issues – Part 2
By Suzin
Stockton
Used by Permission of the Author from the Y2K Health and Detox Center
Used by Permission of the Author from the Y2K Health and Detox Center
JAWBONE CAVITATIONS: Infarction, Infection & Systemic Disease
About seven years ago I made the eye-opening
discovery that my chronic
health problems, which had eluded resolution for many
years, had their origin in – of all places – my jawbone. I would never have
deduced this had it not been for a fortuitous chance finding: the complete
disappearance of bladder problems of one-year duration within days of having an
abscessed tooth removed.
When I reported the “coincidence” to my dentist, he
was incredulous. He shouldn’t have been, for he considered himself to be a
“holistic” practitioner. That same dentist would later, despite his lack of
understanding of focal illness, unknowingly help me to learn more about it by
creating the conditions that triggered the full expression of a long silent
jawbone disease – ischemic osteonecrosis. (a.k.a. cavitations and a dozen or so
other names). This disease is actually quite common, though infrequently
diagnosed, and is perhaps THE most common focal condition in the body. A
“focus” is a walled-off area of concentrated toxins and necrotic (dead) and/or
infected tissue.
Ischemic osteonecrosis (bone death due to poor blood
supply) is a disease of the entire skeleton – i.e., it can affect any bone in
the body. It is best known as a hip condition, and yet it is actually more
common in the jawbone, though unacknowledged as such by mainstream medicine and
dentistry.
A jawbone cavitation is simply a hollow space or
pocket in the bone. It is not readily visible to the eye and often causes no
local discomfort, though it can be the hidden cause of facial pain syndromes
(hence one of its names, NICO – Neuralgia Inducing Cavitational Osteonecrosis).
The chief initiating factor is trauma to the jaw, often brought on by standard
dental treatment.
I’d first encountered the word, “cavitation” many
years ago in the writings of Dr. Hulda Clark. She’d described it in her books
as “a bone infection resulting from an incompletely extracted tooth” – i.e., an
extraction where tissue (bone and ligament) that should be completely extracted
is not thoroughly removed. That description didn’t resonate in me then, despite
the fact that it was exactly what had been silently going on in my jawbone for
many years. I guess I thought if I had an infection in my jaw, I’d know it:
Surely there would be pain, inflammation, tenderness – and my dentist would
find the problem in the course of my routine check-ups. WRONG!
Chronic osteomyelitis (cavitation) of the jawbone is
not characterized by the usual signs of infection (inflammation, redness,
fever, pus) – it most often is a silent condition. And it’s one that dentists
are not trained in school to recognize. In fact, they’re not even taught that
the condition exists. This is a somewhat perturbing state of affairs, for the
jawbone cavitation is not a new disease.
It was described as early as 1848 by Thomas Bond in
the first oral pathology book. He wrote about a jawbone necrosis that existed
independently of abscessed teeth and gums. In 1915, Dr. G.V. Black, the father
of modern dentistry, described the condition as “chronic osteitis.”
Jawbone cavitations are exquisitely described in an
eye-opening book entitled Death and
Dentistry written in 1940 by Martin H. Fischer, medical doctor and
professor of physiology at the University of Cincinnati. Citing the research of
Drs. Frank Billings and E.C. Rosenow (early 1900s), Dr. Fischer speaks of
“infarctions induced of microorganismal emboli” that have broken into the
general circulation from a peripheral focal point in the jaw or tonsils. This
“metastasis” of microorganisms is the cause of a surprising number of
conditions according to Fischer (p.8, 9):
Embolic infection that has struck the heart valves
will be endocarditis; the heart muscle, myocarditis; the pericardium,
pericarditis; if all are struck, it is pancarditis. Involving the skeletal
muscles, the same pathological background will give rise to myositis; when
their tendinous junctions are struck, fibrositis; and when the synovial bursae
are affected, bursitis or tenosynovitis. The process in the joints is
arthritis; and in the nerves and nerve ganglia, neuritis. In the brain, this is
cerebritis, and in its coverings, meningitis.
Death and
Dentistry, by Martin H. Fischer, pages 8-9
Fischer, Martin H., Death and Dentistry. Charles C.
Thomas, LTD:
Springfield, IL, 1940.
Fischer goes on to explain the role of metastatic
infection in gastric and duodenal ulcers, cholecystitis, cystitis, pneumonia,
bronchitis, rheumatism, asthma, pleuritis, nephritis, thyroid disease, herpes,
iritis, poliomyelitis, multiple sclerosis, certain skin disorders, diabetes,
migraines, hypertension and more. He gives case histories and much clinical and
laboratory evidence, including impressive photographs of cross-sections of
infected teeth and microscopy slides.
Although infection in the oral cavity may be a
triggering event in the formation of a cavitation, biopsy of the site typically
shows few, if any, bacteria. It is the toxins produced by these anaerobic
bacteria that are most damaging to the body. However, until local defenses
break down and these toxins gain systemic access, the problem remains localized
and most likely silent.
Symptoms develop when the body burden of toxins
increases to the point that nutritional reserves are depleted, and the system
is no longer able to confine the toxins to their point of origin. They then
travel via blood and lymph channels and through nerve pathways to other areas
of the body.
Toxins create an extremely acidic environment. As
long as the body’s alkaline reserves (primarily calcium and sodium) remain
intact, pH is kept within acceptable limits, homeostasis remains intact, and
the body functions normally. Once alkaline reserves are depleted however,
balance is disrupted. It is not only acid-forming foods (like grains and meat)
so prevalent in the standard American diet, that deplete the alkaline reserves,
but also the bacterial toxins generated at the site of jawbone cavitations.
These toxins create an acid environment and destroy
critical enzyme systems in the body, including enzymes essential for energy
production. The inactivated enzymes are then unable to fulfill their function
as mineral chaperones. The net result is that key minerals, even though present
in the system, become bio-unavailable, for the enzymes needed to activate them
have been destroyed by bacterial toxins.
It is important to understand that such a mineral
“deficiency” is unrelated to mineral intake. It can exist in the face of ample
intake, though insufficient intake certainly compounds the problem. The toxins
responsible for mineral deactivation and breakdown of homeostasis are carried
throughout the system via blood and lymph vessels, tending to settle in areas
of inherent or acquired weakness.
This means that my jawbone cavitations may result in
an entirely different symptom picture than yours, simply because my weaknesses
are different than yours.
The over-acid conditions that result once alkaline
reserves are depleted have many deleterious systemic effects. When the pH of
the blood becomes too acid, its viscosity increases – that is to say it becomes
thicker. Consequently, it does not flow as smoothly through the vessels as it
once did. Clotting anomalies result. A tendency to excessive clotting is very
common in chronic cavitation patients, affecting approximately 80% of them.
Hyper coagulation leads to infarctions in blood vessels.
Jawbone infarctions were spoken of by Dr. Fischer
more than half a century ago. Although the word, “infarct” has come to be
associated with heart attack, the condition is not confined to the large
vessels associated with the heart. Webster defines an “infarct” as “an area of
necrosis in a tissue or organ resulting from obstruction of the local
circulation by a thrombus or embolus.”
Jawbone necrosis does indeed result from impeded
circulation, commonly stemming from trauma to the jawbone. Such trauma is
largely iatrogenic, the result of standard dental treatment. Any large fillings,
crowns, bridges (including the once healthy teeth used as abutments for the
bridge) veneers, endodontic treatment, periodontal scaling, tooth extractions,
injections (particularly of vasoconstrictive anesthetics), placement of toxic
and/or incompatible restorative materials – all of these insults to the jawbone
seriously reduce the blood supply to it. Where blood supply is compromised,
toxins can’t get out, nutrients and oxygen can’t get in.
By the time toxins gain systemic access, alkaline
reserves have become depleted. The blood then becomes hyper viscous, and
infarction can occur. Such infarction tends to occur initially in the small
vessels associated with traumatized bone tissue in the jaw. These infarctions
of the microcirculation, it would appear, are a major factor in the development
and spreading of jawbone cavitations. Fischer understood this years ago when he
wrote of “infarctions induced of microorganismal emboli.”
The dental trauma most often associated with
cavitations is the standard tooth extraction, particularly if it involves the
third molar (or wisdom tooth) sites. Although taught in dental school, it is
not common practice today for the surgeon excavating these teeth to thoroughly
remove the periodontal ligament that attaches tooth to bone.
Once the tooth is removed, this ligament serves no
purpose, and if any part of it is permitted to remain in the jaw, it serves as
a barrier to healing, impeding blood flow and preventing re-growth of bone.
While the extraction site will invariably “heal” shut, the healing is quite
often incomplete, for below the healed-over surface, a pocket or hole has
formed. This hollow space becomes a breeding ground for anaerobic
microorganisms.
It is very possibly these microorganisms that form
the infarction-inducing embolus of which Fischer wrote so many years ago. When
the metabolic waste products of these bacteria interact with chemical toxins
(from restorative materials, anesthetics, etc.) in the oral cavity, the result
is the production of super toxins. The extreme toxicity thus created may well
reduce bacterial population.
Whether or not a cavitation forms following the
standard extraction of a tooth will depend largely upon how much of the
periodontal ligament happens to be removed with the tooth (some portion usually
comes out, even when the surgeon is making no attempt at removal of it) AND the
type of microorganisms which are present at the site.
More damaging than the microorganisms themselves are
the extremely potent toxins they produce. Once these bacterial toxins gain
systemic access, they can do a great deal of harm through inhibition of enzymes
and minerals as described above. The necrosis they produce is actually a
gangrenous condition, which tends to spread to other areas of the jawbone.
Detoxification is a significant challenge at this point and an absolute
impossibility in the face of the continuance of the focal condition (infected
tooth and/or jawbone).
Treatment of choice for jawbone cavitations is
surgical removal of the necrotic and infected bone, for in the presence of such
bone, the conditions that created the infection remain, and blood supply
continues to be impaired. This surgical procedure is a relatively simple one
when done in conjunction with a new extraction.
It is much more difficult where old extraction sites
are concerned. Here the task is complicated by the fact that there has been, up
until very recently, no way to clearly visualize the cavitation site and gain
information about its dimensions and other distinguishing features short of
opening up the site and ‘looking around.’ Even then, the site cannot be viewed
from all angles.
To the trained eye, the panoramic x-ray can reveal
indications of the presence of a cavitation, but not always. Even when it does,
details are often not clearly discernible, and the surgeon is still operating
‘in the blind’ to some degree. The 2-dimensional x-ray image cannot adequately
reflect anomalies in the 3-dimensional jawbone. In some instances, cavitations
can be depicted on x-ray; however, as much as 50% of the bone must be affected
before their presence is apparent.
The MRI, while the ‘gold standard’ for detecting
osteonecrosis of the hip, does not work well with the flat bones of the face.
Tech 99 bone scans are about 70% effective when a special contrast medium is
used. Jawbone cavitations can also be imaged through CT scan, when a spiral
scan is taken from about the middle of the sinus to the bottom of the mandible.
These methods, however, are neither practical nor
cost-effective for use by the dental profession. They expose the patient to the
adverse effects of radiation and require the interpretive services of a
radiologist who is unlikely to recognize jawbone cavitations because he has not
been trained to do so. The aware dentist has long been in need of a reliable
instrument for clearly and safely imaging jawbone cavitations; ideally an
instrument that could be used “in house.” Such an instrument is now available,
due to the unflagging efforts of Bob Jones. The story of his dental drama is interesting,
more dramatic than my own (told in my book, Beyond Amalgam) and worth telling
here.
A decade ago, Bob was a specimen of perfect health –
or so it seemed. He was employed full-time as a commercial airline pilot,
worked part-time as a ski instructor. This avid outdoorsman was slim, trim and
fit. That all changed in 1987 when he was stricken with chronic debilitating
fatigue, muscle atrophy and a neurological condition that baffled specialists.
By 1992, he had become completely disabled, was
wheelchair bound, had lost use of his arms and gained an excessive amount of
weight. While the MDs couldn’t come to agreement on the exact nature of the
problem and finally settled upon a speculative diagnosis of ALS, they were in
agreement on one thing: Bob’s condition was terminal.
They had given him no more than six months to live,
when he stumbled upon an understanding of the source of his problem and a way
to turn it around. His search for solutions led him to the realization that
potent toxins, by-products of standard dental treatments were essentially
poisoning his system. Bob’s symptoms subsided, and his condition dramatically
improved once his diseased bone marrow and “silver” fillings were removed.
Today he is completely mobile and moderately active.
Much of his excess weight has been lost. Bob is quick to point out, however,
that his recovery has not been 100%. At this point in time, chronic cavitation
patients can expect improvement but often not complete cure, owing to the
severity and duration of their condition.
Even before his recovery, Bob set out to develop an
instrument designed to detect jawbone cavitations. Since these lesions
routinely elude detection through standard diagnostic procedures, the need for
an improved imaging device was apparent. As a design engineer with a background
in sonar technology, Bob was convinced from the onset that such an imaging
device could be developed using sonography. Six months after commencing the
arduous task of ‘cleaning out’ his jawbone, Bob had developed the first working
prototype of the CAVITAT™. There would be many design revisions and obstacles
put in his path in the years to follow, but he worked diligently to make his
vision of a perfected CAVITAT™ the reality that it has now become.
The CAVITAT’s proprietary analog to digital circuitry
has been awarded 19 patents. There are 22 additional patents pending on the
flexible circuit receiver and its advanced cross-channel noise suppression
technique. The device is unique in the sonography market in that it is engineered
to show only bone, no soft tissue. All other ultrasound devices do just the
opposite – show tissue but no bone. And, the image they display is
2-dimensional, while the CAVITAT™ displays a 3-dimensional color-coded image.
These colors (green, yellow, red) reflect the degree
of bone loss and necrosis. The 3-D computer images may be rotated so that they
can be viewed from all angles. One image is generated for each of the 32 tooth
sites, and all can be displayed on the screen simultaneously. This allows the
operator to see the overall picture and how one affected site can influence
adjacent ones. Each of the 32 images consists of 64 elements or pixels. These
detailed images are identified as to orientation – “B” for buccal and “D” for
distal.
The new Generation 4 CAVITAT™ differs from its
prototype precursor in many important respects. The resolution has been
increased 800%, making for a much clearer image and enabling detection of
smaller cavitations. The Generation 4 is capable of detecting jawbone defects
down to 1/64 of an inch in diameter.
Bob Jones had introduced a limited number of
Generation 3 CAVITATs to a select number of dentists at the end of 1999. These
were prototype models used for field evaluation. The feedback from the dentists
using them provided the data necessary to make desired improvements. The
software was totally rewritten, and the net result was a user-friendly
state-of-the-art precision instrument. It is this version of the CAVITAT™ that
is now being made available to doctors and dentists to assist in diagnosis of
jawbone cavitations and other bony defects of the jaw.
The significance of this technological break through
cannot be overemphasized. The success of cavitation surgery is dependent upon
many variables. A major one is the extent to which necrotic tissue is removed.
Before the advent of the CAVITAT™, dentists were operating very much in the
blind, unable to see the full extent of the necrosis and therefore unable to
remove all necrotic bone. The result for many patients was poor bone healing,
unchecked spreading of necrotic lesions and consequent need for repeat
surgeries.
While excision of all diseased bone will not
necessarily assure full recovery, it certainly does improve the odds. Most
patients have had jawbone cavitations for a number of years before they are
discovered. Consequently, by the time treatment is initiated, a great deal of
serious damage has been done.
Dr. Fischer had stated in Death and Dentistry, “It is only in the earliest stages of oral
disease that arrest of progressive infection seems possible.” With the
development of the CAVITAT™, early detection is finally possible. It may be our
only hope of putting the reigns on this silent, insidious condition that
appears to have reached epidemic proportions.
While thorough excision of osteonecrotic lesions is
necessary in the treatment of cavitations, for the chronic cavitation patient,
it is often not sufficient. Aggressive detoxification measures are also in
order. These must be tailored to the needs of the individual patient with
regard to his/her specific detoxification capabilities and overall condition.
Nutritional support is also essential – for rebuilding bone, improving
circulation, combating infection, chelating heavy metals.
While surgical treatment of cavitations falls within
the domain of the dental profession, the metastatic infection seeded by these
lesions has systemic consequences that should be of interest to all physicians.
It is therefore imperative that every patient history taken by all physicians
and health care providers include questions about dental treatment. Remember:
Any trauma to the jaw can be the beginning of cavitations.
The high-speed drill routinely used by dentists
cracks enamel, thus allowing bacterial toxins to penetrate the dentine. There
is evidence that such drills cause actual pulp damage. Drilling done then in
preparation of a tooth for routine fillings, crowns and bridges can be damaging
to the jawbone.
Root canals will unquestionably cause cavitations
sooner or later, as will routine extractions (where the socket is not properly
cleaned out, with all necrotic/infected bone removed). The eclectic physician
will not only want to question his patients about these procedures, s/he will
also want to be in a position to diagnose jawbone cavitations, or to refer
patients to a dentist who is able to make such a diagnosis. Once the diagnosis is
made, it is desirable that the dentist and primary physician work together in
instigating a treatment plan and following up with patient.
In working with the chronic cavitation patient, it is
imperative that the entire jawbone be considered and examined – not just the
site(s) of extractions. A mistake that is frequently made is to clean out new
extraction sites, while ignoring old ones. If all necrosis is not removed, it
will spread – and will ultimately re-infect a new extraction site, even one
that was properly cleaned out.
Taking things a step further, it is important to be
aware that the spreading of jawbone cavitations is not confined to edentulous
areas. When the bone beneath an apparently “vital” tooth becomes
affected/infected, blood supply to that tooth is greatly reduced, and it begins
to die. Neither oral exam, nor x-ray evaluation will likely reveal a problem
with such a tooth.
ElectroDermal Screening and muscle testing may also
miss the problem. The patient, however, frequently has a sense of something
being “not quite right” with the tooth. (The chronically sensitive tooth often
is an indication of the presence of jawbone necrosis beneath it) If he or she
insists upon its extraction (usually against the advice of the dentist) and
manages to talk his/her dentist into removing it, that dentist is counseled to
carefully examine the extracted tooth.
Chances are very good that upon drilling into the
pulp chamber, s/he will find that the tooth is dead or dying. This avitality is
reflected by lack of moisture in the pulp chamber, a result of severely
restricted blood flow. I say all of this from personal experience, for three of
my mandibular extractions done in ’99 and ’00 were performed at my insistence
against the initial protestations of my dentist, who fortunately was
open-minded and curious enough to drill open the pulp chambers of the extracted
teeth.
Dentists are taught to save the tooth at all costs.
Frequently, however, the price paid is the systemic health of the patient. Dead
and dying teeth should not remain in the jaw, even if they are causing no acute
distress to the patient. If CAVITAT™ scan of the jawbone shows pronounced
necrosis under a “vital” tooth, please entertain the possibility that the tooth
only appears to be vital, and is, in fact, dying. Healthy teeth don’t grow out
of necrotic bone.
For the chronic cavitation patient, extraction may be
both the beginning and end of his or her health problems. The improperly done
extraction (usually of a wisdom tooth) is frequently the beginning of a problem
which may go undetected for decades, and then only be resolved by the proper
extraction of some, or possibly all, of the remaining teeth, along with removal
of necrotic bone from edentulous areas and aggressive systemic detoxification.
Prevention and early detection are the keys to
avoiding this outcome. Improved imaging capabilities give us the tool for such
early intervention.. The first step in solving the problem, however, is
awareness of it. You have taken that step and are urged to take the next one.
Doctors: Learn to recognize jawbone cavitations and
to either treat them surgically, or refer your patient to a qualified
cavitation surgeon for treatment. Patients: Seek out a dentist familiar with
jawbone pathology: It may be the unsuspected cause of your systemic problems.
Note by
Cancer Tutor: So what does all of this have to do with cancer? Many
cancer cases, as reported by Bill Henderson, can NEVER be cured unless the dental issues discussed above and in the
prior chapter are corrected.
The reason is that cancer is caused by a very special
highly pleomorphic, cell-wall deficient bacteria which can be found inside of
every cancer cell. These microbes block the production of ATP molecules in
cancer cells in several different ways. The microbe which causes cancer can
“hide” in the mouth. This microbe, and the massive toxins caused by dental
procedures, can continually reinfect the cancer.
However, not all cancer patients need the $10,000 or
more procedures to fix their dental issues and finally cure their cancer;
though for those who can afford it, it is certainly a highly recommended
procedure for general health, if not for cancer.
Breast cancer patients are almost certainly in need
of these procedures, and should automatically have them done, but any type of
cancer can be caused by these procedures. See:
Mike Vrentas, who developed the Cellect-Budwig
protocol, has developed a special expertise with regards to breast cancer cases
and is highly recommended as a telephone consultant even if the Cellect-Budwig
protocol is not used.
The individual dentists who do this procedure have
largely gone “underground.” This does not mean they have fled to Mexico, it
simply means that they must “hide in the grass” and avoid too much exposure.
But they can be found.
It should also be emphasized that “biological
dentists” and “holistic dentists” are generally NOT qualified to deal with the dental issues mentioned above!!
Ways to find the very rare dentists who are qualified
was mentioned in the prior chapter.
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Article Topics
- Cavitat
- Death and Dentistry
- Infarction
- Jawbone Cavitations
- Martin H. Fishcer
- Osteonecrosis
- Root Canals
- Suzin Stockton
- Toxins
- Y2K Health and Detox Center
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